De Camilli Lab retweeted
Hancock-Cerutti et al. @PDClab demonstrate that deletion of the autosomal recessive #Parkinsons disease gene VPS13C in a model cell line causes significant perturbations of lysosomal lipid composition and leads to activation of the cGAS-STING pathway bit.ly/3MePOyP
De Camilli Lab retweeted
Congratulations to @PDClab on being featured in the @JCellBiol’s “The Year in Cell Biology: 2021” issue! Specific congratulations to @AGuillenSaman on doing this wonderful work.
De Camilli Lab retweeted
We'd like to give a big shoutout to William Hancock-Cerutti, ASAP's November Network Spotlight!
As an alum of @PDClab at @Yale, his #research helped understand the involvement of protein-mediated lipid transfer between organelles. Learn more about Will: parkinsonsroadmap.org/networ…
De Camilli Lab retweeted
ER-lysosome lipid transfer protein VPS13C/PARK23 prevents aberrant mtDNA-dependent STING signaling biorxiv.org/cgi/content/shor… #biorxiv_cellbio
De Camilli Lab retweeted
We're thrilled to have Pietro De Camilli @PDClab (@YaleNeuro) as a speaker at the upcoming #RUPNeuro Symposium on Neurodegenerative Diseases, April 26-27. See more speakers and register for free here: bit.ly/3pdaBaj
De Camilli Lab retweeted
We’re welcoming 60 new grantees to @ChanZuckerberg’s Neurodegeneration Challenge Network! These researchers are piloting innovative approaches to fighting neurodegenerative diseases like #Alzheimers, #Parkinsons + #ALS czi.co/NeurodegenerationGran…
De Camilli Lab retweeted
from @PDClab, use of DNA-origami nanostructures supports a model where lipids are transferred between ER and plasma membrane through a shuttle mechanism mediated by the synaptotagmin E-Syt1 go.nature.com/2YTUTol (free-to-read at rdcu.be/bKZq2)









