Network toxicology links PFOA to male reproductive disorders via 10 shared genes (AR, ESR1, NOS, BCL2...). Source: doi 10.1186/s12610-026-00328-y Computer predictions plus preliminary cell tests, typically at high doses: relevance to humans unknown.

Bacillus isolates from textile wastewater produce a laccase that decolorizes black, yellow and red dyes at pH 7 and 42 °C. Bacterial enzymes can break down industrial pollutants: a basis for industrial remediation. Source: doi 10.1371/journal.pone.0354516

Full-scale composting plant: 33 PFAS at 4.1 μg/kg in feedstock, 4.0 in finished compost. Composting does not remove PFAS, and oxidizable precursors hide more. Leachate and pit sediments carry them too. Source: doi 10.1016/j.chemosphere.2026.145108

In 1,000+ healthy men, PFAS in seminal plasma (not blood) were associated with longer sperm telomeres; no link with mitochondrial DNA copy number. Association, not cause: the biology is open. Source: doi 10.1016/j.jhazmat.2026.143663

River biofilm exposed to 100 ng/L for two months concentrated PFOS about 15,000-fold and 6:2 FTS about 850-fold. Source: doi 10.1016/j.ecoenv.2026.120850 Accumulation is not removal: it moves PFAS into the base of the food web.

117 Australian tap water samples, 75 PFAS targeted, 65 detected. Levels low (median 1.4 ng/L), but the 6 PFAS regulated by U.S. EPA were a median 11% of the total. A short monitoring list misses most of the mass. Source: doi 10.1016/j.envpol.2026.129260

Fruit flies carry PFAS out of landfills: an estimated 1.49 kg/year across China, rising with warming, especially GenX and 6:2 FTS. Source: doi 10.1016/j.envpol.2026.129264 A landfill is an active PFAS source. Leachate is the bigger exit: destroy PFAS there.

ECHA's committee recommends classifying TFA as reprotoxic and very persistent in water. Before new rules: can we destroy it at scale? At what cost? With what benefit? ECHA could start by funding TFA destruction research. Destroying PFAS is our work at ASPIDIA. #PFAS #TFA

Mice exposed to PFAS for 7 weeks: brain levels similar to other organs, and brain metabolic changes mirrored in plasma, mainly steroid hormones. Source: doi 10.1016/j.envpol.2026.129257 High doses, mouse model: relevance to humans unknown.

In 32-week tests, Pseudomonas degraded the polyacrylamide in polymer-modified bentonite barriers. Source: doi 10.1371/journal.pone.0357812 Bacteria can break down synthetic polymers: the capacity industrial bioremediation builds on to destroy contaminants.

Three estuarine fish species exposed during development to a realistic concentration of PFHxS, with salinity as a second stressor. Source: doi 10.1016/j.cbpc.2026.110703 Experimental models: relevance to humans unknown. Estuaries get PFAS continuously: stop them at the source.

HFPO-TA, a PFOA substitute, caused pparγ-driven fat accumulation in the liver and impaired glucose metabolism in zebrafish. Source: doi 10.1016/j.cbi.2026.112358 High doses, fish model: relevance to humans unknown. Substitutes persist too: destroy them.

In simulated paddy soil, bacteria degraded the bioplastic PHBH almost completely at 35 °C, turning it into methane and CO2. Source: doi 10.1264/jsme2.ME26036 Microbes can destroy a polymer: the principle behind industrial bioremediation. PFAS are the harder target.

To be fair, this is true for almost any company. Most businesses become remarkably profitable once someone else agrees to pay their major costs. The difficult part has always been the bit in between: revenue minus expenses.

PFAS found at 93% of 280 European river sites, 31 countries, 309M people downstream. PFOS and PFOA: rare now. PFHxA, the short-chain replacement: over half the sites. Regulation moved the molecules. It didn't remove the mass. Not destroyed = centuries in water and food chains.