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Perceptio retweeted
Scurvy - Signs, Symptoms & Diagnosis
A 2 year old in the US was diagnosed with a disease from the 1700s
He was brought to the ER because he stopped walking.
No trauma. No fall. He just slowly stopped bearing weight on his legs over 6 weeks until he was crawling instead of walking.
His gums were swollen and bleeding. He had a rash. Pinpoint bleeding under his skin. Pain in both legs. Decreased muscle tone.
He kept his legs in a frog leg position and screamed when anyone touched them.
The doctors suspected everything.
First visit: possible fracture. Orthopedics recommended repeat imaging. Four weeks later he was worse.
Second visit: concern for leukemia. Then concern for nonaccidental trauma. Child protective services was called.
Then the workup began
> CT scan of both legs. No fracture.
> MRI of head and spine. Normal.
> lumbar puncture to rule out Guillain Barre. Normal.
> inflammatory markers. Normal.
> muscle enzymes. Normal.
> heavy metal screening. Negative.
> vitamin D and B12. Normal.
> bone marrow biopsy to rule out cancer. Normal.
> electromyography. Normal.
> muscle biopsy. Normal.
Weeks of testing. Multiple hospital transfers. Invasive procedures on a toddler. His parents watched their child get poked and scanned and biopsied while nobody could tell them what was wrong.
Then someone asked the right question.
What does he eat?
Chocolate milk and graham crackers. That was his entire diet. His mother had mentioned he was a picky eater at the very first visit. Nobody followed up on it.
His vitamin C level came back at less than 0.1 mg per deciliter.
He had scurvy.
The same disease that wiped out entire ship crews during the Age of Exploration. The same disease the British Navy solved in 1795 by issuing lemon juice to sailors.
Scurvy impairs collagen production and compromises blood vessel integrity. That explains the bleeding gums. The rash. The periosteal elevation on MRI. The bone pain. The inability to walk.
Every single symptom pointed to one thing and it took months to get there.
He was started on vitamin C supplements. Within a week he was moving his legs again. At 4 months he was walking and his hemoglobin was normal.
The entire workup could have been avoided with one detailed dietary history at the first visit.
We have MRIs and bone marrow biopsies and genetic panels. But the most powerful diagnostic tool in medicine is still asking a parent what their child eats for dinner.
All that extensive workup could’ve been replaced by a 5 minute conversation about food.
Perceptio retweeted
This man has veins bulging out of his abdomen, fluid filling his belly, and a liver that is barely functioning.
He still hasn’t stopped drinking.
I see this in the ward more often than I’d like to admit.
A known alcoholic. Multiple previous admissions. History of decompensated liver cirrhosis. Ascites drained more than once. Put on diuretics. Counselled every single time. Family begged him to stop.
He was discharged two months ago with strict instructions. No alcohol. Follow up in two weeks. Continue lactulose. Continue spironolactone.
He came back yesterday. Abdomen distended. Prominent dilated veins across the abdominal wall. Caput medusae. The kind you show medical students because you rarely see it this clearly in a textbook.
His liver is so scarred that blood can’t flow through it normally anymore. Portal hypertension forces the blood to find alternate routes. Those veins you see on his abdomen are collaterals. The body’s desperate attempt to bypass a liver that has essentially shut down.
He also has tense ascites. Fluid filling the peritoneal cavity because his liver can no longer produce enough albumin to hold fluid inside the vessels. His serum albumin was 1.8. Normal is 3.5 to 5.5.
We drained 4 litres of fluid from his abdomen yesterday. He’ll need it drained again in weeks. Maybe sooner.
And when I asked him about alcohol his family answered before he could. “He never stopped. Not even for a week.”
This is what alcohol does when your liver has already given up. It’s not a slow decline anymore. Every drink is actively pushing him toward variceal bleeding or hepatic encephalopathy or spontaneous bacterial peritonitis or hepatorenal syndrome. Any one of these can kill him in hours.
The hardest part of medicine isn’t the diagnosis. It isn’t the treatment. It’s watching a patient walk back in with the same condition you treated last time because the one prescription they refused to follow was the only one that mattered.
Stop drinking.
That was the treatment. That was the whole treatment. And he couldn’t do it.
Addiction is a disease. But so is what it leads to. And by the time your abdomen looks like this the damage is not coming back
Perceptio retweeted
Every few weeks someone posts this “doctors used to just check pulse and tongue” nostalgia and it goes viral
Let me tell you what that era actually delivered
Life expectancy in India in 1950 was 35 years. Today it’s 72. That didn’t happen because of tongue examinations.
Here’s what changed.
> heart attack mortality was above 30%. Today it’s under 5%.
> a woman with abdominal pain was told it’s “gas” or “stress.” Today a CT catches appendicitis before it ruptures.
> cancer was found only after it spread. Today screening catches it when it’s still curable.
That stethoscope era you’re glorifying? People died from things we now fix in an afternoon.
The numbers don’t lie.
> breast cancer caught at stage I has 99% survival. Stage IV drops below 30%. Mammography made that gap possible.
> colorectal cancer caught early through colonoscopy has 90%+ survival. Caught late it’s under 15%.
> cervical cancer has been nearly eliminated in countries with routine Pap smears.
None of this. Absolutely none of this. Was possible when the doctor “just checked your pulse.”
Now the “dozen tablets” complaint.
A diabetic today takes metformin an SGLT2 inhibitor a statin and an antihypertensive. That exact combination has been proven across trials involving hundreds of thousands of patients to reduce heart attacks strokes kidney failure and death.
The diabetic 50 years ago? Got told to eat less sugar. Then developed nephropathy retinopathy neuropathy and died of an MI in his 50s.
That’s the era you want back?
The multivitamin point? Actually fair. Most healthy people don’t need them. The evidence for routine multivitamin use in well nourished populations is weak. But that’s a supplement industry problem not a doctor problem.
Now let’s talk about what actually happens when you skip the “costly circus” of a health checkup.
> appendicitis ruptures because nobody imaged it
> a silent MI goes undetected because nobody ran a troponin
> cancer spreads to stage IV because nobody screened for it
> an infection becomes sepsis because nobody ordered a culture
The doctor with just a stethoscope wasn’t practicing some pure form of medicine. He was doing his best with almost nothing. And people died. A lot.
The stethoscope didn’t go away btw. It’s still there. Right next to the ECG the ultrasound the CT and the blood panel that together save more lives in a single hospital shift than a tongue examination did in a decade.
Modern diagnostics isn’t a scam. It’s the reason you’re alive long enough to complain about it on the internet.
There was a time when Doctors used to just check Eyes, Tongue, Pulse and use Stethoscope and do the needful.
Now they need Master Body checkup Plan, Master Body check up Gold Plan, Master Body check up Platinum plan
Then they will prescribe 1 dozen tablets, 3 syrups, few Taqat ke injections to take regularly and then ask again to get Master Body checkup Plan, Master Body check up Gold Plan, Master Body check up Platinum plan
The cycle continues and you hardly no improvement in health.
And what the heck is with Multi Vitamin Tablets?
The question how often Doctor's and their family members go through this costly circus of health checkup ?
Perceptio retweeted
A man in Dallas is alive today because a college student learned CPR in high school.
She learned CPR in high school. Never done it on a real person before. Did it anyway. Kept him alive until paramedics showed up.
Before sharing this. Read what it actually means
> cardiac arrest outside a hospital has about 10% survival rate
> bystander CPR doubles to triples that number
> only 40% of victims in the US receive bystander CPR
> in most developing countries that number is close to zero
The skill takes 30 minutes to learn. Thirty. Minutes.
> where to place your hands
> how deep to compress
> how fast to go
> when to use an AED
That’s it. That’s the entire difference between someone surviving and someone dying on a sidewalk waiting for an ambulance.
Most schools don’t teach it. Most workplaces don’t require it. Most people will live their entire lives without learning the one skill that could keep a stranger’s heart beating.
And every time someone does learn it and uses it we call it a feel good story instead of asking why everyone doesn’t know this already.
Costs nothing. Takes 30 minutes. Doubles survival. Still optional in most of the world.
Emma Dilley, a student at Dallas Baptist University, helped save a stranger's life in downtown Dallas on Tuesday night.
Dilley performed CPR on an unconscious man despite having never done so on a human, before first responders took over medical care.
She learned CPR in high school and was happy that her training came in handy.
CPR classes save lives and good people still exist!
Great job Emma!
Perceptio retweeted
3d reconstruction showing the dilated upper urinary tract and where the ureter is compressed by the menstrual cup.
Perceptio retweeted
A Real Case Where a Simple Menstrual Device Nearly Caused Kidney Failure👇
When urine leaves the kidney, it travels down a narrow tube called the ureter and drains into the bladder.
This process is silent. You never feel it.
But if that flow gets blocked, urine starts backing up.
The kidney swells. Pressure builds. Pain appears.
Doctors call this 𝗵𝘆𝗱𝗿𝗼𝗻𝗲𝗽𝗵𝗿𝗼𝘀𝗶𝘀, and it’s something they take seriously, because untreated, it can damage the kidney.
That’s where this story begins👇
A woman in her early 30s came to the clinic with two unsettling complaints:
𝗜𝗻𝘁𝗲𝗿𝗺𝗶𝘁𝘁𝗲𝗻𝘁 𝗽𝗮𝗶𝗻 on the right side of her lower abdomen and flank
Visible, red-colored urine on several occasions
The pain wasn’t severe. It came and went.
Sometimes it lasted an hour. Sometimes less.
She had no fever.
No burning during urination.
No urgency or frequency.
The symptoms had been going on for six months.
Nothing obvious was wrong
Her medical history was mostly unremarkable.
Years earlier, she had a kidney stone removed from the same side, but that had gone smoothly.
Her blood tests showed:
Normal kidney function
Urine tests showed:
Blood
Mild protein
A camera examination of the bladder showed:
Nothing abnormal
At this point, doctors were stuck.
She had symptoms.
But no explanation.
A CT scan of the urinary tract was ordered.
It finally showed something real:
👉 The right kidney and ureter were dilated, meaning urine was not draining properly.
This explained the pain.
But scans showed:
No stones
No tumors
No clots
No obvious scarring
The bladder itself looked normal.
So why was urine backing up?
Doctors considered two possibilities:
1). A delayed narrowing of the ureter from her stone procedure years earlier
2). A temporary blockage from external pressure
The first explanation didn’t quite fit, strictures usually worsen steadily, not come and go.
The second explanation seemed unlikely.
Until someone looked at the scan again
Carefully.
On the CT images, something else appeared.
An object in the vagina, close to where the ureter enters the bladder.
It wasn’t surgical hardware.
It wasn’t part of the urinary system.
It wasn’t misplaced contraception.
It was symmetrical. Smooth. Soft-looking.
Something the patient had inserted herself.
Something doctors almost overlooked, because it wasn’t expected to cause harm.
When asked more detailed questions about daily habits, the patient mentioned something almost casually:
During the heaviest days of her period, she used a reusable internal menstrual device.
Only for a day or two each month.
She emptied it regularly.
She had never noticed leakage or discomfort.
It seemed irrelevant.
Until the anatomy was considered.
The object pressing near the ureter was a Menstrual Cup.
It had been sitting slightly off-center, positioned just close enough to the point where the ureter drains into the bladder to partially block urine flow.
Not all the time.
Not completely.
Just enough to cause:
Intermittent pain
Back-pressure in the kidney
Episodes of blood in the urine
This condition is called 𝗨𝗿𝗲𝘁𝗲𝗿𝗼𝗵𝘆𝗱𝗿𝗼𝗻𝗲𝗽𝗵𝗿𝗼𝘀𝗶𝘀.
And it was being caused mechanically, from the outside.
Proof came with one simple step
Doctors asked her to stop using the cup.
No medications.
No procedures.
No surgery.
One month later:
The pain was gone
The blood in the urine had stopped
Ultrasound showed complete resolution of the kidney swelling
The kidney was normal again.
Why this case matters:
Menstrual cups are:
Safe
Sustainable
Increasingly popular
And complications are very rare.
But this case shows that:
The ureters run extremely close to the vagina.
Incorrect positioning, size, or anatomy can matter.
Rare complications can look like serious kidney disease.
Only a handful of similar cases have been reported worldwide.
The takeaway:
This is not a warning against menstrual cups.
It’s a reminder that:
Persistent flank pain is never “nothing”
Blood in urine always needs evaluation
Even safe devices can cause problems in rare situations.
And sometimes, the answer isn’t always where you’re looking for it.
Perceptio retweeted
Relieved, she kept using the cream, especially because the child continued to scratch the neck
Days passed.
Then weeks.
And the skin didn’t heal.
It collapsed.
What began as itching turned into an open wound
Perceptio retweeted
A 7 year old developed a simple itch on the neck
No fever. No illness. No injury. Just itching.
Like most parents would, the child’s mother went to a nearby local clinic. She was given a topical cream to apply on the itchy area.
She applied it.
The itching stopped.
Perceptio retweeted
Man presents with jaundice and confusion after taking this HERBAL formula. Diagnosis ??
𝗗𝗼𝗰𝘁𝗼𝗿𝘀 𝗧𝗿𝗲𝗮𝘁𝗲𝗱 𝗢𝗽𝗶𝘂𝗺 𝗢𝘃𝗲𝗿𝗱𝗼𝘀𝗲 𝗯𝘆 𝗕𝗲𝗮𝘁𝗶𝗻𝗴 𝘁𝗵𝗲 𝗣𝗮𝘁𝗶𝗲𝗻𝘁
A historical case where doctors treated opium overdose by beating the patient
In the early 1800s, Laudanum, a powerful mixture of alcohol and opium, was everywhere
It was prescribed for pain, cough, diarrhea…and it was deadly in overdose.
In 1822, a man intentionally swallowed a massive dose of laudanum.
By the time a doctor reached him, hours had passed.
He was deeply unconscious.
Barely breathing.
Completely unresponsive.
Vomiting failed.
Drinks failed.
Irritating the throat with feathers failed.
By all standards of the time, he should have been dead.
That’s when the physician made a desperate decision.
With no internal remedies left, the doctor turned to what he called “high external excitement.”
The patient was repeatedly whipped, on the palms of his hands and the soles of his feet.
When the whipping stopped, the man instantly sank back into deep coma.
When it resumed, he showed pain…then briefly woke.
So they kept going.
For hours
Multiple men took turns beating him to keep him conscious.
If the blows stopped, he collapsed again.
If they resumed, he stirred.
Eventually, after 6–8 hours, the stupor began to lift.
The next day, the man was sleepy, sore, but alive.
Within days, he had fully recovered.
The physician later published the case in 1836, openly admitting:
- He knew of no other recorded example
- No textbook recommended this approach
- It looked cruel, even to doctors of that era
Yet it worked.
Not because whipping “cured” poisoning
but because pain and stimulation kept the nervous system active long enough for the opium to wear off.
Why this matters
This case is disturbing, but important
It shows:
- How deadly opium was before modern antidotes
- How helpless doctors once were
- How survival sometimes came from brutal improvisation, not science
And it’s a reminder that modern medicine didn’t arrive fully formed.
Perceptio retweeted
A Disturbing Attempt to Enter Medical School
A 24 year old man from Jaunpur, Uttar Pradesh, allegedly amputated four toes of his own left foot in an attempt to qualify for MBBS admission through the disability quota, according to reports.
He initially told police that he had been attacked by unknown assailants. A case of attempted murder was registered, and even his family believed the story.
However, investigators found syringes, anesthetic vials, and a cutting machine at the scene. Call records and forensic findings did not support the attack narrative.
The case unraveled after police recovered a diary in which the man described his determination to secure an MBBS seat “at any cost.” Records showed he had earlier applied for a disability certificate, which was rejected.
Police believe that, using his pharmaceutical training, he sedated himself and carried out the amputation.
He is currently under treatment.
unforgettable read
This patient died of a cancer that wasn’t his
When doctors talk about cancer, they’re usually talking about one thing:
your own cells going rogue.
Normal cells follow rules.
Cancer cells don’t.
They grow when they shouldn’t.
They ignore stop signals.
They invade nearby tissue.
They spread to places they don’t belong.
Under the microscope, cancer has a “look” crowded cells, chaotic growth, lots of division, and destruction of normal structures.
So when a 41-year-old man came to the hospital with months of fever, weight loss, cough, and extreme fatigue, doctors were worried.
Scans showed multiple masses:
- In the lungs
- In the liver
- In the adrenal glands
- In lymph nodes throughout the body
This wasn’t one tumor.
This was something spreading.
All signs pointed to aggressive cancer
Biopsies were taken.
Under the microscope, the findings were exactly what doctors fear:
- Cells multiplying uncontrollably
- Invasion into surrounding tissue
- Areas of cell death
- Spread to distant organs
In every way that matters, this behaved like advanced cancer.
But then someone noticed something unsettling.
The cells didn’t look human
Cancer cells come from the body, so they usually resemble the tissue they came from, lung, lymph node, liver, blood.
These cells didn’t.
They were unusually small, even smaller than red blood cells.
They lacked the typical markers of human cells.
They didn’t organize themselves like any known human cancer.
They looked primitive.
Undifferentiated.
Almost…foreign
This raised a terrifying question:
If these cells weren’t behaving like human cells…what were they?
The case becomes a mystery
Doctors tested for everything:
- Common cancers
- Rare cancers
- Fungal infections
- Amoebas
- Exotic parasites
Nothing fit.
Meanwhile, the disease progressed. The masses grew. The patient’s condition worsened.
Experts at the Centers for Disease Control and Prevention (CDC) were consulted. Tissue samples were sent for advanced testing.
And that’s when the case crossed into uncharted territory.
The genetic material inside the “tumor” cells did not belong to the patient.
It wasn’t human.
It belonged to a tapeworm.
Specifically, Hymenolepis nana, one of the most common tapeworms in the world, usually harmless and often symptom free.
But in this man, something unprecedented had happened.
➡️ The tapeworm’s own cells had turned cancerous.
Not a parasite causing cancer.
Not an infection triggering cancer.
A parasite developing cancer inside a human body, then spreading like a malignant tumor.
This had never been documented before.
Why this is a big deal:
Until this case, cancer was considered a disease of the host’s own cells.
This shattered that assumption.
It showed that under extreme conditions, especially severe immune suppression, non-human cells can mutate, grow uncontrollably, invade human tissue, and spread throughout the body like cancer.
In simple terms:
The patient died of cancer that he never developed.
The malignancy that killed him did not arise from his own cells.
It came from something living inside him.
This single case forced doctors to rethink some of the most basic ideas in medicine:
- What cancer actually is
- How infections can behave in weakened bodies
- The role of the immune system as a final line of defense
- And what is biologically possible inside the human body.
wait, why does he look like @ishowspeedsui






















