Computational Biologist researching Alzheimer's Disease progression in the lab of @naomi_habib, @ELSCBrain, @HebrewU , Israel

Joined July 2022
The anti-Israel lunatics are losing their minds because of this video from Hader a village in southern Syria! They‘re asking how it’s possible that Arabs from Syria are asking Israel to annex them to become part of Israel and live under the Israeli ‘apartheid’ regime. Do you want my honest answer, because unlike the uneducated useful idiots in the West the Arabs here know the truth??!!!!! THERE IS NO APARTHEID IN ISRAEL ❗️ Must be so frustrating these days to be anti-Israel 🤣🇮🇱✌🏼
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Thrilled our work is published in @Nature! Analyzing over 1.6M snRNA-seq from 437 brains we uncovered cellular trajectories diverging aging and Alzheimer's disease defined by distinct cell communities. Huge thanks to @naomi_habib and all our collaborators. nature.com/articles/s41586-0…
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(1/5) Excited to share our huge efforts to understand the cellular cascade underlying Alzheimer’s disease progression and untangling disease and aging process. With @naomi_habib, Philip L. De Jager, Vilas Menon, Hyun-Sik Yang and David A. Bennett doi.org/10.1101/2023.03.07.5…
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Gilad Green retweeted
This is what Palestinian CIVILIANS did to a young Israeli civilian on Oct 7th. They paraded their naked body through Gaza as civilians rushed to hit the body with sticks. This is what ‘Free Palestine’ student activists support.
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(1/5) Excited to share our huge efforts to understand the cellular cascade underlying Alzheimer’s disease progression and untangling disease and aging process. With @naomi_habib, Philip L. De Jager, Vilas Menon, Hyun-Sik Yang and David A. Bennett doi.org/10.1101/2023.03.07.5…
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(4/5) Cell populations change along the two trajectories capturing early, transient, and late changes in Alzheimer’s disease, and form coordinated multi-cellular communities specific to each trajectory.
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(5/5) We found multiple populations associated with different aspects of the disease and validated these findings in an independent sample of 684 participants. We suggest a causal AD cascade model placing specific populations in different timepoints along the disease progression
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